Gut bacteria is a major cause of obesity, research says

(NaturalHealth365)  Whether you are fat or thin may be a family trait, but not just through the inheritance of poor eating habits and a tradition of second helpings.  Before you jump to conclusions, this isn’t your typical story about genetics.  If you want to avoid degenerative diseases, I suggest you read this story.

Recent research highlights that your gut microbiome – the trillions of microorganisms living in your digestive tract – plays a pivotal role in determining body weight and overall health.  A study published in Current Gastroenterology Reports explores how gut bacteria, influenced by genetics and lifestyle, are key contributors to obesity and obesity-related disorders.  But here’s the good news: gut microbiota can be modulated.

Nonetheless, this isn’t a free pass to skip healthy eating, though.  The food you consume directly influences which genes are turned on or off, shaping your gut microbiome and metabolic health.  By taking charge of your diet and gut health, you can rewrite the script for your well-being, regardless of genetic predisposition.

Conventionally trained physicians need to learn more about the lifesaving value of gut bacteria

Known collectively as the microbiome, these trillions of live bacteria found in the human body influence everything from your immune system to your likelihood of developing chronic degenerative disease.  These families of bacteria weigh about four pounds and actually outnumber the amount of cells in the body.

The size and weighty influence of these collective bacteria are so significant that some scientists have begun thinking of the microbiome as a “shadow organ” of the human body.  With advanced gene technologies being developed, this extra organ is getting greater attention from scientists worldwide.

New study: How genes are connected to obesity

In one study, scientists from Cornell University joined with researchers at King’s College London to sequence genes of microbes derived from more than 1,000 fecal samples of over 400 pairs of twins.

Twins were used in the research because of their similar gene makeup.  Scientists found that identical twins, whose genes are 100 percent the same, shared greater numbers of specific microbial families in the intestinal tract compared with non-identical twins, who share only half their genes.

These findings provide clear evidence that genes are a major determining factor in the composition of the gut’s microbial makeup.  This microbial composition can influence a host of health factors, including digestion, metabolism and even mood, as well as act as a cause of obesity.

In their published findings, the scientists conclude that identifying bacterial families more prevalent among thin people could hold the key to customized probiotic treatments that exploit healthy traits and lead to weight loss.

Emerging weight loss treatment plans

When transplanted into mice, those microbes found predominantly in thin people were found to actually slow weight gain, suggesting that identification of the exact genetic composition of gut bacteria could hold the key to future weight loss treatments.

Exploiting health-related bacteria families may, one day, be as common of an approach to wellness as exercise and a healthy diet.  These genetic findings may also change how the medical community and the general population approach issues of obesity.

This is truly exciting information – when you consider the fact that more than one-third of United States adults are obese, and millions suffer from obesity-related conditions, including heart disease, stroke, type-2 diabetes, and many forms of cancer – which lead to premature death.

Hopefully, with the help of independent scientists and holistically-minded educators, we’ll achieve critical mass (soon) about the importance of living a healthy lifestyle and implementing safer solutions for the obesity crisis before it’s too late for our society.

Sources for this article include:

NIH.gov
Cell.com
MedicalDaily.com
Cornell.edu
AnnualReviews.org

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